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Type 1 diabetes mellitus (T1DM), also known as insulin-dependent diabetes, is characterized by the autoimmune destruction of pancreatic beta cells, leading to a complete deficiency of insulin and resulting in chronic hyperglycemia (see Figure 1 )

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Some of these, such as c-jun and c-fos, are known to be redox sensitive and can be inactivated by oxidation at critical Cys residues necessary for their DNA-binding activity (1)