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Its functional loss will lead to extracellular glutamate imbalance and activation of extrasynaptic glutamate receptors, increase neuronal excitability, and cause epilepsy (12, 13)
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Interestingly atrial glutathione content is depleted by 58% in NYHA class IV patients compared with NYHA class I patients, which is a drop similar to that found in end-stage failing LV
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