slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells permeability transition redox homeostasis and ferroptosis Glutathione as a Skin Qty:180 Grass Fed Collagen –

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells permeability transition redox homeostasis and ferroptosis Glutathione as a Skin Qty:180 Grass Fed Collagen –

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Mitochondrial glutathione transporter SLC25A40 regulates macrophage cytokine production

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DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated mitochondrial glutathione transport

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Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

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SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells

SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells

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Up-regulated SLC25A39 promotes cell growth and metastasis via regulating ROS production in colorectal cancer

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A hybrid machine learning framework for functional annotation of mitochondrial glutathione transport and metabolism proteins in cancers | BMC Bioinformatics

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Combinatorial GxGxE CRISPR screen identifies SLC25A39 in mitochondrial glutathione transport linking iron homeostasis to OXPHOS

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Slc25a39 and Slc25a40 Expression in Mice with Bile Duct Ligation or Lipopolysaccharide Treatment

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Controlling glutathione entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease

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SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells

SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells

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Mitochondrial glutathione import enables breast cancer metastasis via integrated stress response signaling

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Frontiers

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Development of a mouse model expressing a bifunctional glutathione-synthesizing enzyme to study glutathione limitation in vivo

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SLC25A39 links mitochondrial GSH sensing with iron metabolism: Molecular Cell

SLC25A39 links mitochondrial GSH sensing with iron metabolism: Molecular Cell

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Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis: Molecular Cell

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SLC25A39 identified as a key regulator of hepatocellular carcinoma progression through the mitochondrial ROS–cytochrome c–caspase signaling axis | Cellular & Molecular Biology Letters

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